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Molecular programs induced by heat acclimation confer neuroprotection against TBI and hypoxic insults via cross-tolerance mechanisms

Neuroprotection following prolonged exposure to high ambient temperatures (heat acclimation HA) develops via altered molecular programs such as cross-tolerance Heat Acclimation-Neuroprotection Cross-Tolerance (HANCT). The mechanisms underlying cross-tolerance depend on enhanced “on-demand” protectiv...

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Veröffentlicht in:Front Neurosci
Hauptverfasser: Horowitz, Michal, Umschweif, Gali, Yacobi, Assaf, Shohami, Esther
Format: Artigo
Sprache:Inglês
Veröffentlicht: Frontiers Media S.A. 2015
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Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4516883/
https://ncbi.nlm.nih.gov/pubmed/26283898
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fnins.2015.00256
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