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Phosphorylation of Ser-279/282 and Tyr-265 Positions on Cx43 as Possible Mediators of VEGF-165 Inhibition of Pregnancy-Adapted Ca2+ Burst Function in Ovine Uterine Artery Endothelial Cells
Normal pregnancy requires increased uterine endothelial cell driven vasodilation that is related to increases in sustained Ca2+ signaling via increased connexin 43 (Cx43) gap junction function. Preeclampsia, a hypertensive disorder of pregnancy associated with endothelial dysfunction, is also linked...
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| 出版年: | Mol Cell Endocrinol |
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| 主要な著者: | , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2015
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4516676/ https://ncbi.nlm.nih.gov/pubmed/26033246 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.mce.2015.05.030 |
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