Autophagy-dependent PELI3 degradation inhibits proinflammatory IL1B expression

Lipopolysaccharide (LPS)-induced activation of TLR4 (toll-like receptor 4) is followed by a subsequent overwhelming inflammatory response, a hallmark of the first phase of sepsis. Therefore, counteracting excessive innate immunity by autophagy is important to contribute to the termination of inflamm...

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Detalhes bibliográficos
Publicado no:Autophagy
Main Authors: Giegerich, Annika Klara, Kuchler, Laura, Sha, Lisa Katharina, Knape, Tilo, Heide, Heinrich, Wittig, Ilka, Behrends, Christian, Brüne, Bernhard, von Knethen, Andreas
Formato: Artigo
Idioma:Inglês
Publicado em: Taylor & Francis 2014
Assuntos:
Basic Research Papers
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4502728/
https://ncbi.nlm.nih.gov/pubmed/25483963
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/auto.32178
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