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Fas (CD95) Induces Macrophage Pro-Inflammatory Chemokine Production via a MyD88-dependent, Caspase-independent Pathway
Activation of the prototypical death receptor, Fas (CD95), can induce both caspase-dependent cell death and production of pro-inflammatory chemokines, leading to neutrophil recruitment and end-organ injury. The precise mechanism(s), by which Fas upregulates chemokine production and release, is curre...
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| Pubblicato in: | J Leukoc Biol |
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| Autori principali: | , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2007
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4492281/ https://ncbi.nlm.nih.gov/pubmed/17576821 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1189/jlb.1006652 |
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