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Fas (CD95) Induces Macrophage Pro-Inflammatory Chemokine Production via a MyD88-dependent, Caspase-independent Pathway

Activation of the prototypical death receptor, Fas (CD95), can induce both caspase-dependent cell death and production of pro-inflammatory chemokines, leading to neutrophil recruitment and end-organ injury. The precise mechanism(s), by which Fas upregulates chemokine production and release, is curre...

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Bibliografiske detaljer
Udgivet i:J Leukoc Biol
Main Authors: Altemeier, William A., Zhu, Xiaodong, Berrington, William R., Harlan, John M., Liles, W. Conrad
Format: Artigo
Sprog:Inglês
Udgivet: 2007
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4492281/
https://ncbi.nlm.nih.gov/pubmed/17576821
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1189/jlb.1006652
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