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Induction of Resistance to BRAF Inhibitor Is Associated with the Inability of Spry2 to Inhibit BRAF-V600E Activity in BRAF Mutant Cells

The clinical benefits of oncogenic BRAF inhibitor therapies are limited by the emergence of drug resistance. In this study, we investigated the role of a negative regulator of the MAPK pathway, Spry2, in acquired resistance using BRAF inhibitor-resistant derivatives of the BRAF-V600E melanoma (A375P...

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Bibliografske podrobnosti
izdano v:Biomol Ther (Seoul)
Main Authors: Ahn, Jun-Ho, Han, Byeal-I, Lee, Michael
Format: Artigo
Jezik:Inglês
Izdano: The Korean Society of Applied Pharmacology 2015
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC4489825/
https://ncbi.nlm.nih.gov/pubmed/26157547
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4062/biomolther.2015.007
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