Homocysteine thiolactone and N-homocysteinylated protein induce pro-atherogenic changes in gene expression in human vascular endothelial cells

Genetic or nutritional deficiencies in homocysteine (Hcy) metabolism lead to hyperhomocysteinemia (HHcy) and cause endothelial dysfunction, a hallmark of atherosclerosis. In addition to Hcy, related metabolites accumulate in HHcy but their role in endothelial dysfunction is unknown. Here, we examine...

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Detaylı Bibliyografya
Yayımlandı:Amino Acids
Asıl Yazarlar: Gurda, Dorota, Handschuh, Luiza, Kotkowiak, Weronika, Jakubowski, Hieronim
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Springer Vienna 2015
Konular:
Original Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC4458266/
https://ncbi.nlm.nih.gov/pubmed/25802182
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s00726-015-1956-7
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