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Altered Na(+) Channels Promote Pause-Induced Spontaneous Diastolic Activity in Long QT Syndrome Type 3 Myocytes
Long QT syndrome (LQTS) type 3 (LQT3), typified by the ΔKPQ mutation (LQT3 mutation in which amino acid residues 1505 to 1507 [KPQ] are deleted), is caused by increased sodium entry during the action potential plateau resulting from mutation-altered inactivation of the Na(v)1.5 channel. Although rar...
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| Publicado no: | Circ Res |
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| Main Authors: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2006
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4454351/ https://ncbi.nlm.nih.gov/pubmed/17082480 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/01.RES.0000251305.25604.b0 |
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