Hypoxia-mediated induction of endothelial cell interleukin-1 alpha. An autocrine mechanism promoting expression of leukocyte adhesion molecules on the vessel surface.

Tissue injury that accompanies hypoxemia/reoxygenation shares features with the host response in inflammation, suggesting that cytokines, such as IL-1, may act as mediators in this setting. Human endothelial cells (ECs) subjected to hypoxia (PO2 approximately 12-14 Torr) elaborated IL-1 activity int...

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Publicat a:	J Clin Invest
Autors principals:	Shreeniwas, R, Koga, S, Karakurum, M, Pinsky, D, Kaiser, E, Brett, J, Wolitzky, B A, Norton, C, Plocinski, J, Benjamin, W
Format:	Artigo
Idioma:	Inglês
Publicat:	American Society for Clinical Investigation 1992
Matèries:	Research Article
Accés en línia:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC443387/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1281830/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI116122
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