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The NF-κB regulator Bcl-3 modulates inflammation in contact hypersensitivity reactions in radioresistant cells
Bcl-3 is an atypical member of the IκB family. Bcl-3 functions as a cofactor of p50/NF-κB1 or p52/NF-κB2 homodimers in nuclei, where it modulates NF-κB-regulated transcription in a context-dependent way. Bcl-3 has tumorigenic potential, is critical in host defense of pathogens, and has been reported...
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| Veröffentlicht in: | Eur J Immunol |
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| Hauptverfasser: | , , , , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2015
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4432939/ https://ncbi.nlm.nih.gov/pubmed/25616060 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/eji.201444994 |
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