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The oncoprotein and transcriptional regulator Bcl-3 governs plasticity and pathogenicity of auto-immune T cells

Bcl-3 is an atypical member of the IκB family that modulates transcription in the nucleus via association with p50 (NF-κB1) or p52 (NF-κB2) homodimers. Despite evidence attesting to the overall physiologic importance of Bcl-3, little is known about its cell-specific functions or mechanisms. Here we...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Immunity
Päätekijät: Tang, Wanhu, Wang, Hongshan, Claudio, Estefania, Tassi, Ilaria, Ha, Hye-lin, Saret, Sun, Siebenlist, Ulrich
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2014
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4221807/
https://ncbi.nlm.nih.gov/pubmed/25367572
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.immuni.2014.09.017
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