טוען...
Elevated levels of TWEAK in skeletal muscle promote visceral obesity, insulin resistance, and metabolic dysfunction
Skeletal muscle is responsible for the majority of glucose disposal in body. Impairment in skeletal muscle glucose handling capacity leads to the state of insulin resistance. The TNF-like weak inducer of apoptosis (TWEAK) cytokine has now emerged as a major regulator of skeletal muscle mass and func...
שמור ב:
| הוצא לאור ב: | FASEB J |
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| Main Authors: | , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Federation of American Societies for Experimental Biology
2015
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4422357/ https://ncbi.nlm.nih.gov/pubmed/25466899 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.14-260703 |
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