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The TWEAK-Fn14 dyad is involved in age-associated pathological changes in skeletal muscle
Progressive loss of skeletal muscle mass and strength (sarcopenia) is a major clinical problem in the elderly. Recently, proinflammatory cytokine TWEAK and its receptor Fn14 were identified as key mediators of muscle wasting in various catabolic states. However, the role of the TWEAK-Fn14 pathway in...
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| Autores principales: | , , , , , |
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| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
2014
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4049113/ https://ncbi.nlm.nih.gov/pubmed/24680686 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbrc.2014.03.084 |
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