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The TWEAK-Fn14 dyad is involved in age-associated pathological changes in skeletal muscle

Progressive loss of skeletal muscle mass and strength (sarcopenia) is a major clinical problem in the elderly. Recently, proinflammatory cytokine TWEAK and its receptor Fn14 were identified as key mediators of muscle wasting in various catabolic states. However, the role of the TWEAK-Fn14 pathway in...

詳細記述

保存先:
書誌詳細
主要な著者: Tajrishi, Marjan M., Sato, Shuichi, Shin, Jonghyun, Zheng, Timothy S., Burkly, Linda C., Kumar, Ashok
フォーマット: Artigo
言語:Inglês
出版事項: 2014
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC4049113/
https://ncbi.nlm.nih.gov/pubmed/24680686
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbrc.2014.03.084
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