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Assessment at the Single-Cell Level Identifies Neuronal Glutathione Depletion As Both a Cause and Effect of Ischemia-Reperfusion Oxidative Stress

Oxidative stress contributes to neuronal death in brain ischemia-reperfusion. Tissue levels of the endogenous antioxidant glutathione (GSH) are depleted during ischemia-reperfusion, but it is unknown whether this depletion is a cause or an effect of oxidative stress, and whether it occurs in neurons...

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Bibliografische gegevens
Gepubliceerd in:J Neurosci
Hoofdauteurs: Won, Seok Joon, Kim, Ji-Eun, Cittolin-Santos, Giordano Fabricio, Swanson, Raymond A.
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: Society for Neuroscience 2015
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Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4420782/
https://ncbi.nlm.nih.gov/pubmed/25948264
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4826-14.2015
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