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Control of histone H3 phosphorylation by CaMKIIδ in response to haemodynamic cardiac stress

Heart failure is associated with the reactivation of a fetal cardiac gene programme that has become a hallmark of cardiac hypertrophy and maladaptive ventricular remodelling, yet the mechanisms that regulate this transcriptional reprogramming are not fully understood. Using mice with genetic ablatio...

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Detalles Bibliográficos
Publicado en:J Pathol
Autores principales: Awad, Salma, Al-Haffar, Kamar Mohamed Adib, Marashly, Qussay, Quijada, Pearl, Kunhi, Muhammad, Al-Yacoub, Nadya, Wade, Fallou S, Mohammed, Shamayel Faheem, Al-Dayel, Fouad, Sutherland, George, Assiri, Abdullah, Sussman, Mark, Bers, Donald, Al-Habeeb, Waleed, Poizat, Coralie
Formato: Artigo
Lenguaje:Inglês
Publicado: John Wiley & Sons, Ltd 2015
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC4383650/
https://ncbi.nlm.nih.gov/pubmed/25421395
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/path.4489
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