Cocaine self-administration causes signaling deficits in corticostriatal circuitry that are reversed by BDNF in early withdrawal

Lignende værker

• A Single Brain-Derived Neurotrophic Factor Infusion into the Dorsomedial Prefrontal Cortex Attenuates Cocaine Self-Administration-Induced Phosphorylation of Synapsin in the Nucleus Accumbens during Early Withdrawal
  af: Sun, Wei-Lun, et al.
  Udgivet: (2015)
• Short and long access to cocaine self-administration activates tyrosine phosphatase STEP and attenuates GluN expression but differentially regulates GluA expression in the prefrontal cortex
  af: Sun, Wei-Lun, et al.
  Udgivet: (2013)
• Withdrawal from cocaine self-administration produces long-lasting deficits in orbitofrontal-dependent reversal learning in rats
  af: Calu, Donna J., et al.
  Udgivet: (2007)
• Relapse to Cocaine-seeking after Abstinence Is Regulated by cAMP-dependent Protein Kinase A in the Prefrontal Cortex
  af: Sun, Wei-Lun, et al.
  Udgivet: (2013)
• Oxytocin Reduces Cocaine Seeking and Reverses Chronic Cocaine-Induced Changes in Glutamate Receptor Function
  af: Zhou, Luyi, et al.
  Udgivet: (2014)