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Lack of Glibenclamide Response in a Case of Permanent Neonatal Diabetes Caused by Incomplete Inactivation of Glucokinase
Background: Hypoglycaemic drugs that close the K(ATP) channel have been tested in patients with permanent neonatal diabetes due to glucokinase mutations (PNDM-GCK). From the results obtained, it has been suggested that this treatment may be beneficial in patients carrying GCK mutations with mild kin...
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| Publicado no: | JIMD Rep |
|---|---|
| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Springer Berlin Heidelberg
2015
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4375116/ https://ncbi.nlm.nih.gov/pubmed/25665835 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/8904_2014_383 |
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