Overexpression of N-terminal kinase like gene promotes tumorigenicity of hepatocellular carcinoma by regulating cell cycle progression and cell motility

Amplification and overexpression of CHD1L is one of the most frequent genetic alterations in hepatocellular carcinoma (HCC). Here we found that one of CHD1L downstream targets, NTKL, was frequently upregulated in HCC, which was significantly correlated with vascular invasion (P = 0.012) and poor pro...

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Detalhes bibliográficos
Publicado no:Oncotarget
Main Authors: Wang, Jian, Liu, Ming, Chen, Leilei, Chan, Tim Hon Man, Jiang, Lingxi, Yuan, Yun-Fei, Guan, Xin-Yuan
Formato: Artigo
Idioma:Inglês
Publicado em: Impact Journals LLC 2015
Assuntos:
Research Paper
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4359319/
https://ncbi.nlm.nih.gov/pubmed/25575811
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