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Signaling pathway activation drift during aging: Hutchinson-Gilford Progeria Syndrome fibroblasts are comparable to normal middle-age and old-age cells

For the past several decades, research in understanding the molecular basis of human aging has progressed significantly with the analysis of premature aging syndromes. Progerin, an altered form of lamin A, has been identified as the cause of premature aging in Hutchinson-Gilford Progeria Syndrome (H...

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Detalhes bibliográficos
Publicado no:Aging (Albany NY)
Main Authors: Aliper, Alexander M., Csoka, Antonei Benjamin, Buzdin, Anton, Jetka, Tomasz, Roumiantsev, Sergey, Moskalev, Alexey, Zhavoronkov, Alex
Formato: Artigo
Idioma:Inglês
Publicado em: Impact Journals LLC 2015
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4350323/
https://ncbi.nlm.nih.gov/pubmed/25587796
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