Platelet Count Mediates the Contribution of a Genetic Variant in LRRC16A to ARDS Risk

BACKGROUND: Platelets are believed to be critical in pulmonary-origin ARDS as mediators of endothelial damage through their interactions with fibrinogen and multiple signal transduction pathways. A prior meta-analysis identified five loci for platelet count (PLT): BAD, LRRC16A, CD36, JMJD1C, and SLM...

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Detalhes bibliográficos
Publicado no:Chest
Main Authors: Wei, Yongyue, Wang, Zhaoxi, Su, Li, Chen, Feng, Tejera, Paula, Bajwa, Ednan K., Wurfel, Mark M., Lin, Xihong, Christiani, David C.
Formato: Artigo
Idioma:Inglês
Publicado em: American College of Chest Physicians 2015
Assuntos:
Original Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4347530/
https://ncbi.nlm.nih.gov/pubmed/25254322
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1378/chest.14-1246
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