Macrophage apoAI protects against dyslipidemia-induced dermatitis and atherosclerosis without affecting HDL

Tissue cholesterol accumulation, macrophage infiltration, and inflammation are features of atherosclerosis and some forms of dermatitis. HDL and its main protein, apoAI, are acceptors of excess cholesterol from macrophages; this process inhibits tissue inflammation. Recent epidemiologic and clinical...

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Dades bibliogràfiques
Publicat a:J Lipid Res
Autors principals: Tavori, Hagai, Su, Yan Ru, Yancey, Patricia G., Giunzioni, Ilaria, Wilhelm, Ashley J., Blakemore, John L., Zabalawi, Manal, Linton, MacRae F., Sorci-Thomas, Mary G., Fazio, Sergio
Format: Artigo
Idioma:Inglês
Publicat: The American Society for Biochemistry and Molecular Biology 2015
Matèries:
Research Articles
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4340310/
https://ncbi.nlm.nih.gov/pubmed/25593328
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1194/jlr.M056408
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