Macrophage apoAI protects against dyslipidemia-induced dermatitis and atherosclerosis without affecting HDL

Tissue cholesterol accumulation, macrophage infiltration, and inflammation are features of atherosclerosis and some forms of dermatitis. HDL and its main protein, apoAI, are acceptors of excess cholesterol from macrophages; this process inhibits tissue inflammation. Recent epidemiologic and clinical...

Disgrifiad llawn

Wedi'i Gadw mewn:
Manylion Llyfryddiaeth
Cyhoeddwyd yn:J Lipid Res
Prif Awduron: Tavori, Hagai, Su, Yan Ru, Yancey, Patricia G., Giunzioni, Ilaria, Wilhelm, Ashley J., Blakemore, John L., Zabalawi, Manal, Linton, MacRae F., Sorci-Thomas, Mary G., Fazio, Sergio
Fformat: Artigo
Iaith:Inglês
Cyhoeddwyd: The American Society for Biochemistry and Molecular Biology 2015
Pynciau:
Research Articles
Mynediad Ar-lein:https://ncbi.nlm.nih.gov/pmc/articles/PMC4340310/
https://ncbi.nlm.nih.gov/pubmed/25593328
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1194/jlr.M056408
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