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Phosphorylation of Shox2 Is Required for Its Function to Control Sinoatrial Node Formation
BACKGROUND: Inactivation of Shox2, a member of the short‐stature homeobox gene family, leads to defective development of multiple organs and embryonic lethality as a result of cardiovascular defects, including bradycardia and severe hypoplastic sinoatrial node (SAN) and sinus valves, in mice. It has...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | J Am Heart Assoc |
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| Κύριοι συγγραφείς: | , , , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
Blackwell Publishing Ltd
2014
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4309068/ https://ncbi.nlm.nih.gov/pubmed/24847033 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/JAHA.114.000796 |
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