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Hydrogen sulfide epigenetically attenuates homocysteine-induced mitochondrial toxicity mediated through NMDA receptor in mouse brain endothelial (bEnd3) cells()
Previously we have showed that homocysteine (Hcy) caused oxidative stress and altered mitochondrial function. Hydrogen sulphide (H(2)S) has potent anti-inflammatory, anti-oxidative and anti-apoptotic effects. Therefore, in the present study we examined whether H(2)S ameliorates Hcy-induced mitochond...
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| Udgivet i: | J Cell Physiol |
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| Main Authors: | , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
2015
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4305357/ https://ncbi.nlm.nih.gov/pubmed/25056869 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/jcp.24722 |
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