Delayed cutaneous wound closure in HO-2 deficient mice despite normal HO-1 expression

Impaired wound healing can lead to scarring, and aesthetical and functional problems. The cytoprotective haem oxygenase (HO) enzymes degrade haem into iron, biliverdin and carbon monoxide. HO-1 deficient mice suffer from chronic inflammatory stress and delayed cutaneous wound healing, while corneal...

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Detalhes bibliográficos
Publicado no:J Cell Mol Med
Main Authors: Lundvig, Ditte M S, Scharstuhl, Alwin, Cremers, Niels A J, Pennings, Sebastiaan W C, te Paske, Jeroen, van Rheden, René, van Run-van Breda, Coby, Regan, Raymond F, Russel, Frans G M, Carels, Carine E, Maltha, Jaap C, Wagener, Frank A D T G
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Publishing Ltd 2014
Assuntos:
Original Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4302653/
https://ncbi.nlm.nih.gov/pubmed/25224969
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.12389
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