Toll-like receptor 3 activation is required for normal skin barrier repair following UV damage

Ultraviolet (UV) damage to the skin leads to the release of noncoding RNA (ncRNA) from necrotic keratinocytes that activates toll-like receptor 3 (TLR3). This release of ncRNA triggers inflammation in the skin following UV damage. Recently, TLR3 activation was also shown to aid wound repair and incr...

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Bibliographic Details
Published in:J Invest Dermatol
Main Authors: Borkowski, Andrew W., Kuo, I-Hsin, Bernard, Jamie J., Yoshida, Takeshi, Williams, Michael R., Hung, Nai-Jung, Yu, Benjamin D., Beck, Lisa A., Gallo, Richard L.
Format: Artigo
Language:Inglês
Published: 2014
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Article
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC4289479/
https://ncbi.nlm.nih.gov/pubmed/25118157
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/jid.2014.354
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