Redox dysregulation, neuroinflammation, and NMDA receptor hypofunction: a “central hub” in schizophrenia pathophysiology ?

Accumulating evidence points to altered GABAergic parvalbumin-expressing interneurons and impaired myelin/axonal integrity in schizophrenia. Both findings could be due to abnormal neurodevelopmental trajectories, affecting local neuronal networks and long-range synchrony and leading to cognitive def...

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Detalles Bibliográficos
Publicado en:Schizophr Res
Main Authors: Steullet, P., Cabungcal, J.H., Monin, A., Dwir, D., O’Donnell, P., Cuenod, M., Do, K.Q.
Formato: Artigo
Idioma:Inglês
Publicado: 2014
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Article
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC4282982/
https://ncbi.nlm.nih.gov/pubmed/25000913
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.schres.2014.06.021
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