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The genome-wide transcriptional response to neonatal hyperoxia identifies Ahr as a key regulator
Premature infants requiring supplemental oxygen are at increased risk for developing bronchopulmonary dysplasia (BPD). Rodent models involving neonatal exposure to excessive oxygen concentrations (hyperoxia) have helped to identify mechanisms of BPD-associated pathology. Genome-wide assessments of t...
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| Publicat a: | Am J Physiol Lung Cell Mol Physiol |
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| Autors principals: | , , , , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Physiological Society
2014
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4280157/ https://ncbi.nlm.nih.gov/pubmed/25150061 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00200.2014 |
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