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The genome-wide transcriptional response to neonatal hyperoxia identifies Ahr as a key regulator

Premature infants requiring supplemental oxygen are at increased risk for developing bronchopulmonary dysplasia (BPD). Rodent models involving neonatal exposure to excessive oxygen concentrations (hyperoxia) have helped to identify mechanisms of BPD-associated pathology. Genome-wide assessments of t...

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Detalhes bibliográficos
Publicado no:Am J Physiol Lung Cell Mol Physiol
Main Authors: Bhattacharya, Soumyaroop, Zhou, Zhongyang, Yee, Min, Chu, Chin-Yi, Lopez, Ashley M., Lunger, Valerie A., Solleti, Siva Kumar, Resseguie, Emily, Buczynski, Bradley, Mariani, Thomas J., O'Reilly, Michael A.
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2014
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4280157/
https://ncbi.nlm.nih.gov/pubmed/25150061
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00200.2014
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