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Netrin-1 Abrogates Ischemia Reperfusion-induced Cardiac Mitochondrial Dysfunction via Nitric Oxide-dependent Attenuation of NOX4 Activation and Recoupling of NOS
Despite an established role of mitochondrial dysfunction in cardiac ischemia reperfusion (I/R) injury, the upstream activators have remained incompletely defined. We have recently identified an innovative role of exogenously applied netrin-1 in cardioprotection, which is mediated by increased nitric...
Tallennettuna:
Julkaisussa: | J Mol Cell Cardiol |
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Päätekijät: | , , , |
Aineistotyyppi: | Artigo |
Kieli: | Inglês |
Julkaistu: |
2014
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Aiheet: | |
Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4268247/ https://ncbi.nlm.nih.gov/pubmed/25066694 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2014.07.005 |
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