Therapeutic targeting of GSK3β enhances the Nrf2 antioxidant response and confers hepatic cytoprotection in hepatitis C

OBJECTIVE: Impaired adaptive response to oxidative injuries is a fundamental mechanism central to the pathogenesis of chronic hepatitis C (CHC). Glycogen synthase kinase (GSK) 3β is an indispensable regulator of the oxidative stress response. However, the exact role of GSK3β in CHC is uncertain and...

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Gepubliceerd in:Gut
Hoofdauteurs: Jiang, Yongfang, Bao, Hui, Ge, Yan, Tang, Wei, Cheng, Du, Luo, Kaizhong, Gong, Guozhong, Gong, Rujun
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: 2014
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Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4263291/
https://ncbi.nlm.nih.gov/pubmed/24811996
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1136/gutjnl-2013-306043
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