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Opposite regulation of inhibitory synaptic plasticity by α and β subunits of Ca(2+)/calmodulin-dependent protein kinase II

Induction of several forms of synaptic plasticity, a cellular basis for learning and memory, depends on the activation of Ca(2+)/calmodulin (CaM)-dependent protein kinase II (CaMKII). CaMKII acts as a holoenzyme consisting of α and β subunits (α- and βCaMKII). However, it remains elusive how the sub...

詳細記述

保存先:
書誌詳細
出版年:J Physiol
主要な著者: Nagasaki, Nobuhiro, Hirano, Tomoo, Kawaguchi, Shin-ya
フォーマット: Artigo
言語:Inglês
出版事項: BlackWell Publishing Ltd 2014
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC4259534/
https://ncbi.nlm.nih.gov/pubmed/25217378
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2014.280230
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