Enhanced glycemic responsiveness to epinephrine in insulin-dependent diabetes mellitus is the result of the inability to secrete insulin. Augmented insulin secretion normally limits the glycemic, but not the lipolytic or ketogenic, response to epinephrine in humans.

Eitemau Tebyg

• Enhanced glycemic responsiveness to epinephrine in insulin-dependent diabetes mellitus is the result of the inability to secrete insulin. Augmented insulin secretion normally limits the glycemic, but not the ipolytic or ketogenic, response to epinephrine in humans
  Cyhoeddwyd: (1985)
• Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man.
  gan: Rosen, S G, et al.
  Cyhoeddwyd: (1984)
• Epinephrine plasma thresholds for lipolytic effects in man: measurements of fatty acid transport with [l-13C]palmitic acid.
  gan: Galster, A D, et al.
  Cyhoeddwyd: (1981)
• Impaired secretion of epinephrine in response to insulin among hypophysectomized dogs.
  gan: Wurtman, R J, et al.
  Cyhoeddwyd: (1968)
• Mechanisms of postprandial glucose counterregulation in man. Physiologic roles of glucagon and epinephrine vis-a-vis insulin in the prevention of hypoglycemia late after glucose ingestion.
  gan: Tse, T F, et al.
  Cyhoeddwyd: (1983)