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APA (7. basım) Alıntı
Berk, M. A., Clutter, W. E., Skor, D., Shah, S. D., Gingerich, R. P., Parvin, C. A., & Cryer, P. E. (1985). Enhanced glycemic responsiveness to epinephrine in insulin-dependent diabetes mellitus is the result of the inability to secrete insulin. Augmented insulin secretion normally limits the glycemic, but not the lipolytic or ketogenic, response to epinephrine in humans. J Clin Invest.
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Chicago Style (17. basım) Atıf
Berk, M A., W E. Clutter, D. Skor, S D. Shah, R P. Gingerich, C A. Parvin, ve P E. Cryer. "Enhanced Glycemic Responsiveness to Epinephrine in Insulin-dependent Diabetes Mellitus Is the Result of the Inability to Secrete Insulin. Augmented Insulin Secretion Normally Limits the Glycemic, but Not the Lipolytic or Ketogenic, Response to Epinephrine in Humans."
J Clin Invest 1985.
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MLA (9th ed.) Atıf
Berk, M A., et al. "Enhanced Glycemic Responsiveness to Epinephrine in Insulin-dependent Diabetes Mellitus Is the Result of the Inability to Secrete Insulin. Augmented Insulin Secretion Normally Limits the Glycemic, but Not the Lipolytic or Ketogenic, Response to Epinephrine in Humans."
J Clin Invest, 1985.
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