Cita APA

Berk, M. A., Clutter, W. E., Skor, D., Shah, S. D., Gingerich, R. P., Parvin, C. A., & Cryer, P. E. (1985). Enhanced glycemic responsiveness to epinephrine in insulin-dependent diabetes mellitus is the result of the inability to secrete insulin. Augmented insulin secretion normally limits the glycemic, but not the lipolytic or ketogenic, response to epinephrine in humans.

Chicago Style Citation

Berk, M A., W E. Clutter, D. Skor, S D. Shah, R P. Gingerich, C A. Parvin, i P E. Cryer. Enhanced Glycemic Responsiveness to Epinephrine in Insulin-dependent Diabetes Mellitus Is the Result of the Inability to Secrete Insulin. Augmented Insulin Secretion Normally Limits the Glycemic, but Not the Lipolytic or Ketogenic, Response to Epinephrine in Humans. 1985.

Cita MLA

Berk, M A., et al. Enhanced Glycemic Responsiveness to Epinephrine in Insulin-dependent Diabetes Mellitus Is the Result of the Inability to Secrete Insulin. Augmented Insulin Secretion Normally Limits the Glycemic, but Not the Lipolytic or Ketogenic, Response to Epinephrine in Humans. 1985.