Epstein-Barr Virus Glycoprotein gB and gHgL Can Mediate Fusion and Entry in trans, and Heat Can Act as a Partial Surrogate for gHgL and Trigger a Conformational Change in gB

Lignende værker

• Fusion of Epstein-Barr Virus with Epithelial Cells Can Be Triggered by αvβ5 in Addition to αvβ6 and αvβ8, and Integrin Binding Triggers a Conformational Change in Glycoproteins gHgL
  af: Chesnokova, Liudmila S., et al.
  Udgivet: (2011)
• Fusion of epithelial cells by Epstein–Barr virus proteins is triggered by binding of viral glycoproteins gHgL to integrins αvβ6 or αvβ8
  af: Chesnokova, Liudmila S., et al.
  Udgivet: (2009)
• Use of gHgL for Attachment of Epstein-Barr Virus to Epithelial Cells Compromises Infection
  af: Borza, Corina M., et al.
  Udgivet: (2004)
• Epstein-Barr virus infection mechanisms
  af: Chesnokova, Liudmila S., et al.
  Udgivet: (2014)
• Structural basis for Epstein–Barr virus host cell tropism mediated by gp42 and gHgL entry glycoproteins
  af: Sathiyamoorthy, Karthik, et al.
  Udgivet: (2016)