טוען...
Dysregulation of antiviral helicase pathways in systemic lupus erythematosus
In the autoimmune disease systemic lupus erythematosus (SLE), our normal antiviral defenses are inappropriately activated, resulting in over-activity of the type I interferon (IFN) pathway. This increased activity of the type I IFN pathway is an important primary pathogenic factor in the disease. Em...
שמור ב:
| הוצא לאור ב: | Front Genet |
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| Main Authors: | , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Frontiers Media S.A.
2014
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4243696/ https://ncbi.nlm.nih.gov/pubmed/25505487 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fgene.2014.00418 |
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