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5-Azacytidine acts directly on both erythroid precursors and progenitors to increase production of fetal hemoglobin.

The effect of 5-azacytidine on erythroid precursors and progenitors was studied in nine patients with sickle cell anemia or severe thalassemia. Each patient received the drug intravenously for 5 or 7 d. 5-Azacytidine caused a four- to sixfold increase in gamma-messenger RNA concentration in bone mar...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:J Clin Invest
Päätekijät: Humphries, R K, Dover, G, Young, N S, Moore, J G, Charache, S, Ley, T, Nienhuis, A W
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society for Clinical Investigation 1985
Aiheet:
Linkit:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC423530/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2579100/
https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI111731
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