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Increased glomerular thromboxane synthesis as a possible cause of proteinuria in experimental nephrosis.

Altered glomerular metabolism of arachidonic acid (AA) has already been demonstrated in experimental nephrotoxic nephritis. The enhanced synthesis of thromboxane A2 (TxA2) in isolated glomeruli that has been found may mediate changes in renal hemodynamics. The objectives of this investigation were:...

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Detalhes bibliográficos
Main Authors: Remuzzi, G, Imberti, L, Rossini, M, Morelli, C, Carminati, C, Cattaneo, G M, Bertani, T
Formato: Artigo
Idioma:Inglês
Publicado em: 1985
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC423412/
https://ncbi.nlm.nih.gov/pubmed/4038407
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