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Increase in cytosolic Ca(2+) produced by hypoxia and other depolarizing stimuli activates a non-selective cation channel in chemoreceptor cells of rat carotid body
The current model of O(2) sensing by carotid body chemoreceptor (glomus) cells is that hypoxia inhibits the outward K(+) current and causes cell depolarization, Ca(2+) influx via voltage-dependent Ca(2+) channels and a rise in intracellular [Ca(2+)] ([Ca(2+)](i)). Here we show that hypoxia (<5% O...
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| Publicado no: | J Physiol |
|---|---|
| Main Authors: | , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
BlackWell Publishing Ltd
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4230773/ https://ncbi.nlm.nih.gov/pubmed/24591572 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2013.266957 |
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