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Synaptic and Cognitive Improvements by Inhibition of 2-AG Metabolism Are through Upregulation of MicroRNA-188-3p in a Mouse Model of Alzheimer's Disease

Abnormal accumulation of β-amyloid (Aβ) is the major neuropathological hallmark of Alzheimer's disease (AD). However, the mechanisms underlying aberrant Aβ formation in AD remain unclear. We showed previously that inhibition of monoacylglycerol lipase (MAGL), the primary enzyme that metabolizes...

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Détails bibliographiques
Publié dans:J Neurosci
Auteurs principaux: Zhang, Jian, Hu, Mei, Teng, Zhaoqian, Tang, Ya-Ping, Chen, Chu
Format: Artigo
Langue:Inglês
Publié: Society for Neuroscience 2014
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Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC4220026/
https://ncbi.nlm.nih.gov/pubmed/25378159
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1165-14.2014
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