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Synaptic and Cognitive Improvements by Inhibition of 2-AG Metabolism Are through Upregulation of MicroRNA-188-3p in a Mouse Model of Alzheimer's Disease

Abnormal accumulation of β-amyloid (Aβ) is the major neuropathological hallmark of Alzheimer's disease (AD). However, the mechanisms underlying aberrant Aβ formation in AD remain unclear. We showed previously that inhibition of monoacylglycerol lipase (MAGL), the primary enzyme that metabolizes...

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Detalles Bibliográficos
Publicado en:J Neurosci
Autores principales: Zhang, Jian, Hu, Mei, Teng, Zhaoqian, Tang, Ya-Ping, Chen, Chu
Formato: Artigo
Lenguaje:Inglês
Publicado: Society for Neuroscience 2014
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC4220026/
https://ncbi.nlm.nih.gov/pubmed/25378159
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1165-14.2014
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