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Interfacial binding and aggregation of lamin A tail domains associated with Hutchinson-Gilford progeria syndrome
Hutchinson-Gilford progeria syndrome is a premature aging disorder associated with the expression of Δ50 lamin A (Δ50LA), a mutant form of the nuclear structural protein lamin A (LA). Δ50LA is missing 50 amino acids from the tail domain and retains a C-terminal farnesyl group that is cleaved from th...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4212650/ https://ncbi.nlm.nih.gov/pubmed/25194277 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bpc.2014.08.005 |
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