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Liver metastases induce reversible hepatic B cell dysfunction mediated by Gr-1+CD11b+ myeloid cells

LM escape immune surveillance, in part, as a result of the expansion of CD11b+MC, which alter the intrahepatic microenvironment to promote tumor tolerance. HBC make up a significant proportion of liver lymphocytes and appear to delay tumor progression; however, their significance in the setting of L...

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Detalhes bibliográficos
Main Authors: Thorn, Mitchell, Point, Gary R., Burga, Rachel A., Nguyen, Cang T., Joseph Espat, N., Katz, Steven C.
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Leukocyte Biology 2014
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4197561/
https://ncbi.nlm.nih.gov/pubmed/25085111
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1189/jlb.3A0114-012RR
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