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Th17 can regulate silica-induced lung inflammation through an IL-1β-dependent mechanism
Silicosis is an occupational lung disease caused by the inhalation of silica dust and characterized by lung inflammation and fibrosis. Interleukin (IL)-1β is induced by silica and functions as the key pro-inflammatory cytokine in this process. The Th17 response, which is induced by IL-1β, has been r...
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| Główni autorzy: | , , , , , , , , |
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| Format: | Artigo |
| Język: | Inglês |
| Wydane: |
Blackwell Publishing Ltd
2014
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| Hasła przedmiotowe: | |
| Dostęp online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4196653/ https://ncbi.nlm.nih.gov/pubmed/25091058 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.12341 |
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