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Differential nuclear localization of complexes may underlie in vivo intrabody efficacy in Huntington's disease
Intrabodies offer attractive options for manipulating the protein misfolding that triggers neurodegenerative diseases. In Huntington's disease, where the expanded polyglutamine tract in the extreme N-terminal region of huntingtin exon1 misfolds, two lead intrabodies have been selected against a...
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| Hoofdauteurs: | , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Oxford University Press
2014
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4191446/ https://ncbi.nlm.nih.gov/pubmed/25301961 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/protein/gzu041 |
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