ERK activation is required for CCK-mediated pancreatic adaptive growth in mice

High levels of cholecystokinin (CCK) can stimulate pancreatic adaptive growth in which mature acinar cells divide, leading to enhanced pancreatic mass with parallel increases in protein, DNA, RNA, and digestive enzyme content. Prolonged release of CCK can be induced by feeding trypsin inhibitor (TI)...

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Main Authors: Holtz, Bryan J., Lodewyk, Kevin B., Sebolt-Leopold, Judith S., Ernst, Stephen A., Williams, John A.
Format: Artigo
Jezik:Inglês
Izdano: American Physiological Society 2014
Teme:
Pancreas
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC4187068/
https://ncbi.nlm.nih.gov/pubmed/25104499
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpgi.00163.2014
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