PIN3 duplication may be partially responsible for TP53 haploinsufficiency

BACKGROUND: Previously we have suggested that cancer cells develop a mechanism(s) which allows for either: silencing of the wild-type TP53 transcription, degradation of the wild-type TP53 mRNA, or selective overproduction of the mutated TP53 mRNA, which is the subject of this article. Sequencing of...

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Autors principals: Winiecka-Klimek, Marta, Szybka, Malgorzata, Rieske, Piotr, Piaskowski, Sylwester, Bienkowski, Michal, Walczak, Maciej, Pacholczyk, Marcin, Rostkowski, Michal, Zieba, Jolanta, Banaszczyk, Mateusz, Hulas-Bigoszewska, Krystyna, Peciak, Joanna, Pawliczak, Rafal, Stoczynska-Fidelus, Ewelina
Format: Artigo
Idioma:Inglês
Publicat: BioMed Central 2014
Matèries:
Research Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC4176858/
https://ncbi.nlm.nih.gov/pubmed/25223755
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2407-14-669
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