Reciprocal modulations between p53 and Tat of human immunodeficiency virus type 1.
Infection by human immunodeficiency virus type 1 (HIV-1) causes acquired immunodeficiency syndrome (AIDS) after a long clinical latency. This disease is associated with a spectrum of cancers. Here we report that wild-type p53 is a potent suppressor of Tat, a major transactivator of HIV-1. Reciprocal...
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| Publicado no: | Proc Natl Acad Sci U S A |
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| Principais autores: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
1995
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC41714/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7777531/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.92.12.5461 |
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