CHOLECYSTOKININ RECEPTOR ANTAGONIST HALTS PROGRESSION OF PANCREATIC CANCER PRECURSOR LESIONS AND FIBROSIS IN MICE

OBJECTIVES: Exogenous administration of cholecystokinin (CCK) induces hypertrophy and hyperplasia of the pancreas with an increase in DNA content. We hypothesized that endogenous CCK is involved with the malignant progression of pancreatic intraepithelial neoplasia (PanIN) lesions and the fibrosis a...

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Detalhes bibliográficos
Main Authors: Smith, Jill P., Cooper, Timothy K., McGovern, Christopher O., Gilius, Evan L., Zhong, Qing, Liao, Jiangang, Molinolo, Alfredo A., Gutkind, J. Silvio, Matters, Gail L.
Formato: Artigo
Idioma:Inglês
Publicado em: 2014
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4161619/
https://ncbi.nlm.nih.gov/pubmed/25058882
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1097/MPA.0000000000000194
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