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Myeloid Kruppel-like Factor 2 deficiency exacerbates neurological dysfunction and neuroinflammation in a murine model of multiple sclerosis
Cells of the innate immune system are important mediators of multiple sclerosis (MS). We have previously identified Kruppel-like Factor 2 (KLF2) as a critical negative regulator of myeloid activation in the setting of bacterial infection and sepsis, but the role of myeloid KLF2 in MS has not been in...
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| Huvudupphovsmän: | , , , , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
2014
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4152407/ https://ncbi.nlm.nih.gov/pubmed/25052583 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jneuroim.2014.06.023 |
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