Neuroprotection after traumatic brain injury in heat-acclimated mice involves induced neurogenesis and activation of angiotensin receptor type 2 signaling

Long-term exposure of mice to mild heat (34°C±1°C) confers neuroprotection against traumatic brain injury (TBI); however, the underling mechanisms are not fully understood. Heat acclimation (HA) increases hypothalamic angiotensin II receptor type 2 (AT(2)) expression and hypothalamic neurogenesis. A...

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Autores principales: Umschweif, Gali, Shabashov, Dalia, Alexandrovich, Alexander G, Trembovler, Victoria, Horowitz, Michal, Shohami, Esther
Formato: Artigo
Lenguaje:Inglês
Publicado: Nature Publishing Group 2014
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Original Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC4126099/
https://ncbi.nlm.nih.gov/pubmed/24849663
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/jcbfm.2014.93
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